Dictionary

All 170 gene variations of mybody x at a glance

Doppelhelix aus Glas auf hellem Grund mit grünen und hellblauen Lichtreflexen

Search, filter, and discover all genetic analyses - from nutrition and nutrient requirements to sports, sleep, and metabolism.

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Maximum oxygen uptake is the single most important measure of endurance performance - and one of the strongest predictors of life expectancy overall. PGC-1alpha controls the formation of new mitochondria, VEGFA the formation of new blood vessels in muscle, ACE vascular regulation, and ADRB2 the response to adrenaline.

Genes analyzed5
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

People respond very differently to the same endurance training - the range goes from barely measurable improvement to an increase of over 40 percent. The analysis shows where you need to focus if your body responds more slowly.

What the variants mean

Certain variants are associated in studies with a stronger training adaptation, others with a weaker one. Those who respond less strongly need more volume or more targeted intensity control to achieve the same results.

What you can do

With lower responsiveness, high-intensity intervals often work better than basic endurance training alone because they provide a stronger stimulus for mitochondrial formation. With good responsiveness, a high proportion of low-intensity endurance sessions is usually enough.

Frequently asked questions

Can I have my VO2max measured?
Yes, through spiroergometry or approximately through field tests. Genetic analysis shows your predisposition; the test shows your actual level.

Am I untalented if I have an unfavorable variant?
No. Fitness level has a much greater influence on VO2max than genetic predisposition. It only determines how quickly you progress.

Genes analyzed

ACEADRB2PPARAPPARGC1AVEGFA

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Alcohol is broken down in two steps. First, alcohol dehydrogenase converts it into acetaldehyde - a substance that is significantly more toxic than alcohol itself and is responsible for headaches, nausea, and facial flushing. In the second step, the aldehyde dehydrogenase ALDH2 neutralizes this intermediate. If either step is slowed, acetaldehyde builds up.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

People who break down acetaldehyde slowly are under significantly greater strain - even with small amounts and even when they don’t notice any symptoms. This explains why people can have very different tolerances despite drinking the same amount.

What the variants mean

The slow ALDH2 variant is common in East Asia and causes the well-known facial flushing after drinking alcohol. In Europe, variants of the ADH genes are more relevant, as they alter the rate of breakdown in the first step.

What you can do

When breakdown is slowed, the most effective measure is to reduce the amount - breakdown cannot be sped up. Staying hydrated, eating while drinking, and having alcohol-free days also reduce the strain. The liver enzymes GPT and Gamma-GT are suitable for monitoring over time.

Frequently asked questions

What causes facial flushing after drinking alcohol?
It’s caused by acetaldehyde building up when its breakdown is slowed. It’s a visible warning sign of increased strain, not a harmless effect.

Can I speed up alcohol breakdown?
No. Neither coffee, exercise, nor home remedies change the rate of breakdown. Only time helps - and drinking less.

Genes analyzed

ADH1BADH1CALDH2

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PPAR-alpha is a regulatory protein that controls fat breakdown in the liver and muscles. It is activated by polyunsaturated fatty acids and then regulates genes that burn fatty acids for energy. The strength of this response varies genetically.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

This value explains why some people benefit greatly from switching to plant oils and fish, while others see hardly any change.

What the variants mean

For certain variants, studies show a significantly stronger response in blood lipid levels to increased intake of polyunsaturated fatty acids. This mainly affects triglycerides.

What you can do

If you have a strong response variant, it’s worth consistently focusing on rapeseed and flaxseed oils, walnuts, and oily fish. If your response is weaker, exercise and reducing refined carbohydrates are usually more effective levers.

Frequently asked questions

Does this replace measuring my blood lipid levels?
No. Genetic analysis shows your predisposition, while blood tests show your current state. They complement each other - together, they provide a complete picture.

Genes analyzed

PPARA

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Tendons and ligaments consist mostly of collagen. COL5A1 influences its fiber structure, MMP3 the remodeling and breakdown of connective tissue, and GDF5 the development of joints and tendons. Together, they shape how stiff or flexible the tissue is and how quickly it recovers after exertion.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Achilles tendon, ACL, and rotator cuff injuries are among the most common reasons for long training breaks. Knowing your predisposition lets you train proactively rather than treat injuries reactively.

What the variants mean

Certain variants have been linked in studies to a higher frequency of tendon and ligament injuries among physically active people.

What you can do

With an unfavorable predisposition, the most effective measures are eccentric strength training for tendons, thorough warm-ups, gradually increasing training volume, and adequate recovery. A good intake of vitamin C and protein also supports collagen formation.

Frequently asked questions

Does this mean I should avoid certain sports?
No. It means planning increases in training load more carefully and taking preparation more seriously. Injuries are almost always caused by increasing too quickly, not by the sport itself.

Genes analyzed

COL5A1GDF5MMP3

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Telomeres are protective caps at the ends of chromosomes that shorten with each cell division. Once they fall below a critical length, the cell stops dividing. TERC is a component of telomerase, the enzyme that can lengthen telomeres. Variants in TERC are associated with different telomere lengths.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Telomere length is considered one marker of biological, as opposed to chronological, age. It can be influenced - smoking, chronic stress, lack of sleep, and excess weight cause it to shorten faster.

What the variants mean

Certain variants are associated with shorter telomeres on average. Genetics plays a substantial role, but lifestyle does too and, unlike genetic predisposition, can be influenced.

What you can do

Observational studies identify quitting smoking, regular exercise, a Mediterranean diet, adequate sleep, and active stress reduction as telomere-protective. Supplements promising to protect telomeres lack reliable evidence.

Frequently asked questions

Can I lengthen my telomeres?
It is difficult to control how much they lengthen, but the rate at which they shorten can be influenced. Quitting smoking, exercise, and sleep are the best-supported factors.

Does this predict my life expectancy?
No. Telomere length is a statistical marker at the population level and does not allow conclusions about any individual person.

Genes analyzed

TERC

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Blood sugar regulation is a finely tuned system involving insulin release, insulin action, and sugar release from the liver. TCF7L2 affects insulin release from the pancreas, SLC30A8 affects zinc transport in insulin-producing cells, MTNR1B affects nighttime insulin regulation via melatonin, and G6PC2 affects fasting blood sugar.

Genes analyzed8
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

These variants explain why some people respond to a high-carbohydrate meal with significantly sharper blood sugar spikes than others, even when eating the same thing. Knowing this makes it possible to structure carbohydrate intake strategically.

What the variants mean

Certain variants are associated with higher fasting blood sugar levels and a delayed insulin response. MTNR1B is particularly interesting: in carriers of this variant, the blood sugar response to late meals is significantly less favorable.

What you can do

Effective approaches include high-fiber carbohydrate sources instead of fast sugars, protein and vegetables before the carbohydrate component of a meal, exercise after eating, and - for the MTNR1B variant - moving the last meal earlier, to the early evening. HbA1c in the blood can be used to monitor progress.

Frequently asked questions

Does an unfavorable variant mean I'll get diabetes?
No. This analysis is not a diagnostic test and does not predict disease. It describes how your metabolism responds to carbohydrates - and this influence is much smaller than that of weight, exercise, and diet.

Why is eating late less favorable with MTNR1B?
Melatonin rises in the evening and suppresses insulin release more strongly in carriers of this variant. The same meal then leads to higher blood sugar levels late in the evening than at midday.

Genes analyzed

ADCY5DGKBG6PC2GCKRIRS1MTNR1BSLC30A8TCF7L2

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The internal clock is controlled by a small core in the brain that generates an approximately 24-hour rhythm through clock genes such as CLOCK and NPAS2. This rhythm determines sleep and wakefulness, as well as body temperature, hormone release, and metabolic activity throughout the day.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

A large part of the population lives against their own internal clock - work schedules rarely align with chronotype. The resulting shift between biological and social time measurably affects sleep quality and metabolism.

What the variants mean

Certain variants are associated with a later tendency to sleep, later sleep onset, and, in studies, higher calorie intake in the evening.

What you can do

You can't retrain your chronotype, but you can mitigate it: bright light in the morning shifts your internal clock earlier, while dim lighting and avoiding screens in the evening prevent it from shifting later. If you're strongly an evening type, avoid scheduling important tasks for the early morning. In combination with the MTNR1B variant, having your last meal earlier is also advisable.

Frequently asked questions

Can I make myself a morning person?
Only to a limited extent. Chronotype is largely determined by genetics and changes mainly with age. A shift of one to two hours is realistic with consistent light management.

What is social jet lag?
The difference between your sleep timing on days off and workdays. The greater it is, the more you live against your own internal clock.

Genes analyzed

CLOCKNPAS2

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The body has no active way to excrete iron—the only way to regulate it is through absorption in the gut. This is controlled by the hormone hepcidin. TMPRSS6 influences its production, while HFE affects signal transmission. Variants in both genes shift the balance toward greater or lower absorption.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Iron is the only nutrient for which both deficiency and excess matter. The analysis shows which way your predisposition leans—and whether iron supplements make sense for you or should be used with caution.

What the variants mean

Variants in TMPRSS6 are often associated with lower iron and ferritin levels. Certain HFE variants are associated with increased iron absorption.

What you can do

Genetic analysis does not replace blood testing—the ferritin level remains key, supplemented by transferrin saturation and CRP. If you tend to absorb more iron, only take iron supplements after testing.

Frequently asked questions

Should I supplement with iron if I have an unfavorable variant?
Only after testing. Taking iron without a confirmed deficiency is not advisable, and is not recommended if you are predisposed to increased absorption.

Does this replace a medical evaluation?
No. This analysis provides lifestyle and nutrition information. Abnormal blood iron levels should always be checked by a doctor.

Genes analyzed

HFETMPRSS6

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Being overweight is associated with persistent, low-grade inflammation - fat tissue itself releases messenger substances such as TNF-alpha and interleukin-6. The strength of this response is partly determined by genetics. The analyzed variants affect both the production of these messenger substances and the level of CRP in the blood.

Genes analyzed6
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Low-grade inflammation is one of the links between abdominal fat, insulin resistance, and vascular aging. People with a pronounced predisposition to react this way benefit especially from an anti-inflammatory diet and good sleep quality.

What the variants mean

Certain variants are associated with higher baseline levels of inflammatory markers. This does not mean there is inflammation - it describes how strongly the system responds when irritated.

What you can do

Omega-3 fatty acids, plenty of vegetables and fiber, reducing alcohol and highly processed foods, getting enough sleep, and reducing abdominal fat are effective. Measuring CRP in the blood alongside these measures shows whether they are working.

Frequently asked questions

How does this relate to the CRP level in a blood test?
Genes describe predisposition; CRP in the blood reflects the current state. People with a pronounced predisposition should monitor their blood levels over time.

Genes analyzed

CRPHNF1AIL6IL6RTNFTNFA

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Lipoprotein lipase sits on blood vessel walls and breaks down fats in the blood so muscle and fat cells can absorb them. PPAR-alpha simultaneously regulates the genes in the muscles that burn fatty acids. Together, they help determine how early and how strongly the body switches to fat during endurance exercise.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

People who metabolize fat less efficiently at low intensity tire earlier during long sessions and benefit from a different training plan.

What the variants mean

Certain variants are associated with more efficient fat metabolism and more favorable HDL and triglyceride levels.

What you can do

Fat burning can be trained: long, easy sessions at a low heart rate measurably raise the point at which the body switches to carbohydrates. This effect is independent of genetic predisposition.

Frequently asked questions

Do I burn the most fat in the fat-burning heart rate zone?
Proportionally, yes; absolutely, not necessarily. Weight loss depends on total energy expenditure, not the percentage of fat burned.

Genes analyzed

LPLPPARA

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The MTHFR enzyme converts folate into its active form, 5-methyltetrahydrofolate. This is needed to break down homocysteine and provide methyl groups that regulate numerous metabolic processes. Certain variants significantly reduce enzyme activity—by up to 70 percent in the most common form.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

MTHFR variants are very common in Europe. People with a variant break down homocysteine more slowly and benefit more from the active form of folate than from conventional folic acid.

What the variants mean

When enzyme activity is reduced, homocysteine levels in the blood rise measurably if there isn’t enough folate, vitamin B12, and vitamin B6.

What you can do

Good sources include leafy greens, legumes, and whole grains. If a variant is confirmed, the already activated form, 5-MTHF, is often preferred. Measuring homocysteine in the blood at the same time is useful—it shows whether your intake is sufficient.

Frequently asked questions

Is an MTHFR variant dangerous?
No. It is very common and is not a disease in itself. It becomes relevant only if you also don’t get enough folate, B12, or B6.

What does this mean if I’m trying to conceive?
Having an adequate folate supply before conception is important regardless of the variant. If enzyme activity is reduced, the active form is more often recommended—ask your doctor for advice.

Genes analyzed

MTHFR

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Celiac disease can develop only if certain HLA markers are present. Without these markers, celiac disease is virtually ruled out. The analysis is therefore mainly useful for ruling it out—it reliably tells you what cannot be the cause.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

About 30 to 40 percent of the population have these markers, but only a fraction actually develop celiac disease. The value of the analysis therefore lies in ruling it out: if you don’t have these markers, there’s no need to investigate this cause further.

What the variants mean

Without these markers, celiac disease is very unlikely. With them, it is possible, but by no means certain—the vast majority of people with these markers have no symptoms.

What you can do

Important: This analysis is not a diagnosis. If you have persistent symptoms after eating gluten, you should see a doctor for testing—through blood antibody tests and, if necessary, a tissue sample. Crucially, you must not stop eating gluten before testing, or the results will be unusable.

Frequently asked questions

Do I have celiac disease if I have these markers?
No. These markers are a prerequisite, not a diagnosis. Only a small proportion of people with these markers actually develop celiac disease—it must be diagnosed by a doctor.

Should I avoid gluten as a precaution?
No. Avoiding it without a diagnosis has no proven benefit and makes later medical evaluation impossible, because the antibodies will no longer be detectable.

Genes analyzed

DQA1

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HDL collects excess cholesterol from the tissues and carries it back to the liver. ABCA1 loads cholesterol onto HDL particles, LCAT packages it, CETP exchanges it for fats from other particles, and LIPC and LIPG break down HDL. Each of these steps varies genetically.

Genes analyzed9
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

HDL is the measure that most stubbornly resists attempts to change it. The analysis shows how much is genetic—and where the remaining levers lie.

What the variants mean

Certain CETP variants are associated with significantly higher HDL levels, while variants in ABCA1 and LIPC are associated with lower levels. The genetic influence on HDL is stronger than for most other blood fats.

What you can do

Regular endurance exercise, not smoking, and weight loss are effective. Medications specifically intended to raise HDL have shown no benefit in studies, so the focus should be on LDL and triglycerides.

Frequently asked questions

My HDL is low despite exercising—what could be the reason?
Often largely genetic. HDL is strongly determined by genetics. In any case, low LDL and low triglycerides are more important for vascular health.

Genes analyzed

ABCA1CETPGALNT2LCATLIPCLIPGLRP1PLTPPPARA

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Endurance training changes the heart: the left ventricle becomes larger and more powerful, resting heart rate decreases, and stroke volume increases. CREB1 is a regulatory protein involved in this adaptation; ACE influences blood vessel regulation and heart growth through the renin-angiotensin system.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The heart’s ability to adapt partly determines how far endurance performance can improve—and is also a key factor in long-term heart health.

What the variants mean

Certain ACE variants have been associated in studies with greater heart adaptation to endurance training, while others are more often linked to benefits from strength and power training.

What you can do

For heart adaptation, long, steady sessions at low intensity provide the most effective stimulus, complemented by a few intense intervals. Resting heart rate is a good marker of progress: it measurably decreases as adaptation improves.

Frequently asked questions

Is a low resting heart rate always a good thing?
In trained people, it is a sign of good adaptation. A sudden, pronounced change in resting heart rate unrelated to training should be checked by a doctor.

Genes analyzed

ACECREB1

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Neuromedin B is a messenger molecule involved in regulating hunger and satiety in the brain. Together with BDNF, which influences the development of appetite-regulating nerve cells, it shapes how clearly the satiety signal is received and how quickly appetite returns after eating.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

A weak satiety signal is one of the most common unrecognized reasons diets fail. It explains why some people feel full after the same portion while others do not.

What the variants mean

Certain variants are associated in studies with larger portions, more frequent snacking, and a stronger return of hunger after eating.

What you can do

Effective approaches include high-volume, low-calorie meals with plenty of vegetables, enough protein at every meal, and eating slowly. Getting enough sleep also matters: sleep deprivation significantly shifts hunger and satiety hormones, regardless of genetics.

Frequently asked questions

Why don't I feel full after eating?
Alongside diet composition and eating speed, the genetic makeup of satiety signals also plays a role. Meals rich in protein and fiber are the most effective way to compensate.

Genes analyzed

BDNFNMB

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Adiponectin is produced by fat tissue itself and has anti-inflammatory and insulin-sensitizing effects. Remarkably, the more belly fat there is, the less adiponectin is produced - a self-reinforcing cycle. The ADIPOQ gene partly determines the level of production.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Adiponectin links belly fat, insulin sensitivity, and low-grade inflammation. Those with an unfavorable genetic predisposition benefit more than average from reducing belly fat - even a few kilograms can measurably improve hormone levels.

What the variants mean

Certain variants are associated with lower adiponectin levels. Studies show this mainly as weaker insulin sensitivity at the same body weight.

What you can do

Effective approaches include targeted belly fat reduction, regular aerobic activity, and a high-fiber diet. All three increase adiponectin levels regardless of genetic makeup.

Frequently asked questions

Can I directly influence adiponectin levels?
Not through supplements, but lifestyle can make a difference. Losing visceral belly fat is the most effective approach.

Genes analyzed

ADIPOQ

Related analysis topics

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WNK1 is a regulatory protein in the kidneys that controls the reabsorption of sodium and potassium. It sits at a control point that determines how much potassium is excreted and how much is retained - indirectly affecting blood pressure as well.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Potassium is sodium’s natural counterpart. Adequate potassium intake measurably lowers blood pressure, but it is rarely given special attention in the diet.

What the variants mean

Certain variants are associated with altered potassium excretion and, in studies, slightly elevated blood pressure.

What you can do

Vegetables, legumes, potatoes, nuts and fruit are rich in potassium. People with an unfavorable variant benefit especially from a plant-focused diet. If you have impaired kidney function, potassium intake should be managed by a doctor.

Frequently asked questions

Should I take potassium supplements?
Usually not. You can easily meet your needs through vegetables and fruit, and supplements can be dangerous if you have impaired kidney function.

Genes analyzed

WNK1

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Bone is living tissue that is continuously built up and broken down throughout life. COL1A1 encodes type 1 collagen, the basic framework of bone, while ESR1 encodes the estrogen receptor, through which hormones regulate bone remodeling. The other variants affect signaling pathways that regulate the balance between bone formation and breakdown.

Genes analyzed9
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Peak bone mass is built up by around age 30, after which it declines. Knowing early on that you have an unfavorable predisposition lets you take action during the period when it can make the biggest difference.

What the variants mean

Certain variants are associated with, on average, lower bone density in large studies. The effect of individual variants is small, but measurable when combined.

What you can do

Strength training and weight-bearing exercise, adequate calcium, vitamin D and vitamin K, and sufficient protein are effective. Avoiding smoking and drinking alcohol in moderation provide additional protection. Bone density itself can only be measured with a DXA scan.

Frequently asked questions

Does the analysis predict whether I will develop osteoporosis?
No. It describes a predisposition in bone metabolism and is not a diagnostic test. Bone density must be assessed with a DXA scan, and diagnosis requires a medical evaluation.

What helps most to build strong bones?
Impact. Strength training and weight-bearing activities such as jumping or running provide the strongest stimulus for bone formation - far stronger than any supplement.

Genes analyzed

AKAP11C6ORF97COL1A1DCDC5ESR1GPR177SP7TNFRSF11AZBTB40

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About 95 percent of caffeine is broken down in the liver by the CYP1A2 enzyme. Depending on the variant, this happens quickly or slowly. In slow metabolizers, caffeine stays in the blood significantly longer - so an afternoon coffee can affect you into the night without you noticing.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

This is one of the most practically useful analyses, because you can put the findings into action right away: if you metabolize caffeine slowly, move your caffeine cutoff earlier and sleep measurably better.

What the variants mean

Fast metabolizers tolerate more caffeine and are more likely to benefit from its positive effects in studies. In slow metabolizers, the effects last longer, and higher amounts are associated with less favorable cardiovascular responses.

What you can do

If you metabolize caffeine slowly, it's best to have your last caffeinated drink by early afternoon at the latest and limit your daily intake. Keep in mind: green and black tea, cola, and energy drinks count too.

Frequently asked questions

How long does caffeine stay in the body?
The half-life averages about five hours, and is significantly longer in slow metabolizers. So after eight hours, a substantial amount is still active.

Why do I fall asleep easily despite drinking coffee?
Falling asleep and sleep quality are two different things. Caffeine mainly reduces deep sleep - you don't notice it when falling asleep, but the next morning.

Genes analyzed

CYP1A2

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All mammals stop producing lactase after weaning - humans originally did too. A few thousand years ago, a variant emerged in Europe in the regulatory region of MCM6 that keeps the lactase gene switched on permanently. Most people in Northern Europe carry this variant; worldwide, it is the exception.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Bloating, abdominal pain and diarrhea after dairy products are often attributed to other causes. Genetic analysis can determine in one step whether you have the genetic predisposition to produce lactase at all.

What the variants mean

With the variant associated with no lactase persistence, enzyme activity decreases over the course of life. Symptoms often appear only in adulthood - and become more severe with age.

What you can do

Without lactase persistence, lactose-free products, aged hard cheese and yogurt are usually well tolerated because most of the lactose has been broken down. Important: Avoiding dairy products must not put your calcium intake at risk - mineral water, green vegetables and fortified plant-based drinks can make up for it.

Frequently asked questions

I tolerate milk, but have the unfavorable variant - how is that possible?
For many people, the remaining lactase activity is enough to handle small amounts. Symptoms depend on the amount, gut flora and accompanying meal.

Is this the same as a milk allergy?
No. Lactose intolerance is caused by a missing enzyme, while a milk allergy occurs when the immune system reacts to milk protein. They have different causes and consequences.

Genes analyzed

MCM6

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Only a small part of LDL levels is determined by the cholesterol on your plate. What matters is how much the liver produces itself and how efficiently it removes LDL from the blood. LDLR encodes this very receptor, which clears LDL, APOB encodes the recognition protein on the LDL particle, and ABCG5 and ABCG8 regulate the absorption of cholesterol from the intestine.

Genes analyzed10
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

This analysis sheds light on one of the most persistent debates in nutrition advice: some people respond strongly to dietary cholesterol, while most barely respond at all. ABCG5 and ABCG8 distinguish these two groups.

What the variants mean

Certain variants are associated with higher LDL levels on average. Variants in ABCG5 and ABCG8 also cause a significantly stronger response to dietary cholesterol.

What you can do

Effective for everyone: replace saturated fats with unsaturated fats, increase fiber, and optimize weight and exercise. If you are particularly sensitive to dietary cholesterol, it may also be worth limiting cholesterol-rich foods. Your actual LDL level should be checked in a blood test - genes only describe your predisposition.

Frequently asked questions

Do I have to give up eggs?
Not for most people - dietary cholesterol has less impact on blood cholesterol levels than once thought. However, people with certain variants in ABCG5 and ABCG8 have a significantly stronger response.

Does the analysis replace cholesterol testing?
No. Genes show predisposition; blood test results show the current state. The measured value is what matters for assessing risk.

Genes analyzed

ABCG5/8ANGPTL3APOBAPOEGCKRHLAIL6RLDLRPPP1R3BTIMD4

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Wanting sweets isn't just a matter of willpower. SLC2A2 also transports glucose into brain cells that monitor the body's sugar needs - studies link variants of this gene to higher sugar consumption. ADRA2A affects how strongly people crave sweets via the nervous system.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Knowing that you're predisposed to crave sweets more strongly can help you respond with structure instead of self-reproach - for example, by not keeping sweets at home in the first place, rather than fighting the urge every day.

What the variants mean

In observational studies, carriers of certain variants measurably consume more sugar without consciously controlling it.

What you can do

Effective strategies include keeping blood sugar levels stable with high-protein breakfasts, eating fiber with every meal, and consistently keeping sugar out of sight. Not keeping sweets in stock beats willpower.

Frequently asked questions

Is a strong craving for sweets genetic?
This shouldn't be called addiction. It's about a stronger or weaker craving that can be managed effectively through dietary structure.

Genes analyzed

ADRA2ASLC2A2

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ACTN3 produces a structural protein found exclusively in fast-twitch muscle fibers - the fibers responsible for short, powerful movements. About 18 percent of people carry two inactive copies and produce none of this protein. In them, a related protein takes over, shifting the muscle more toward endurance.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Few analyses can be applied so directly to training. It explains why some people gain strength quickly with weight training but struggle with running - and vice versa.

What the variants mean

Among elite sprinters and strength athletes, the active variant is significantly overrepresented; among endurance athletes, the inactive variant is. For recreational athletes, the effect is small, but useful as a general indication.

What you can do

With a strength-oriented profile, heavy sets with few repetitions and longer rests work best. With an endurance-oriented profile, higher repetitions, shorter rests, and more training volume are more effective. Both can be trained—the genetic predisposition only determines where you make faster progress.

Frequently asked questions

Does the endurance variant mean I can't build muscle?
Yes, absolutely. It may take a little longer for some people, and it works better with different stimuli. The variant describes a tendency, not a limit.

Is ACTN3 used in elite sports?
It is the most extensively studied sports variant, but it is not suitable for talent selection. The effects of training, motivation, and technique are many times greater.

Genes analyzed

ACTN3

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CHRNA3 encodes a component of the nicotine receptor in the brain. Variants in this gene are among the best-documented genetic influences on smoking behavior. They affect how rewarding nicotine feels, how many cigarettes are smoked each day, and how severe withdrawal symptoms are.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Knowing that quitting is genetically harder for you can change how you approach it—with support rather than on your own, and without seeing a relapse as a personal failure.

What the variants mean

In studies, carriers of certain variants smoke more cigarettes per day on average and are less likely to quit successfully without support.

What you can do

For those with a strong predisposition, structured programs, medical support, and, if appropriate, nicotine replacement are much more effective than trying to quit on your own. This increases the success rate severalfold.

Frequently asked questions

Is smoking genetic?
Whether someone starts smoking is shaped largely by social factors. How strong the addiction becomes and how difficult quitting is, however, are significantly influenced by genetics.

Genes analyzed

CHRNA3

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Plant-based omega-3 sources such as flaxseed and rapeseed oil provide alpha-linolenic acid. For the body to use it, it must convert it into EPA and DHA—a process that depends on FADS enzymes. This conversion is inefficient to begin with, and certain variants reduce the rate even further.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

For people with an unfavorable variant, the recommendation to eat flaxseed oil instead of fish is practically ineffective. They need EPA and DHA directly—from oily sea fish or algal oil.

What the variants mean

For certain variants, the conversion rate of plant-based omega-3 into EPA and DHA is significantly lower. Studies show correspondingly lower EPA and DHA levels in the blood.

What you can do

If conversion is limited, two servings of fatty fish per week or an algal oil supplement are the direct route. It is also worth reducing omega-6-rich oils such as sunflower oil, as they compete for the same enzymes.

Frequently asked questions

Is flaxseed oil enough as an omega-3 source?
With a good conversion rate, partly; with limited conversion, hardly at all. Even under the most favorable conditions, the conversion of plant-based omega-3 into EPA and DHA is only in the single-digit percentage range.

What is the alternative for vegans?
Algal oil provides EPA and DHA directly, so it does not depend on the conversion rate.

Genes analyzed

FADS1FADS1-2-3

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Lean body mass is not just a matter of training. IL15RA affects the action of a messenger that regulates muscle growth; TRHR is associated with the amount of lean mass, and MCT-1 with lactate transport out of muscle cells and therefore with capacity for intense exercise.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Muscle mass is about much more than looks: it determines basal metabolic rate and insulin sensitivity, and supports independence as you age. If you know that building muscle is harder for you, plan your training and protein intake more consistently.

What the variants mean

Certain variants have been associated in studies with greater lean body mass and a stronger response to strength training.

What you can do

Regardless of your genetic predisposition, the basics are the same: progressively increase the load, do two to three strength-training sessions per week, and consume 1.6 to 2.0 grams of protein per kilogram of body weight. If your genetic predisposition is less favorable, consistency over the years pays off most.

Frequently asked questions

How much protein do I need to build muscle?
About 1.6 to 2.0 grams per kilogram of body weight daily. Studies show no additional benefit from consuming more.

Genes analyzed

IL15RAMCT-1TRHR

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APOA2 is one of the best-known examples of a gene-diet interaction. The gene influences how the body processes saturated fatty acids. For some people, a high-fat diet has a greater effect on body weight than it does for others, even with the same calorie intake.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The question of whether a low-fat or higher-fat diet is a better fit has been answered with sweeping generalizations for decades. APOA2 is one of the few markers that can support an individual answer.

What the variants mean

Studies show that people with the sensitive variant have a significantly higher BMI when they consume large amounts of saturated fat. With low intake, the difference disappears completely - the predisposition only has an effect when challenged by diet.

What you can do

If you have the sensitive variant, it's worth limiting saturated fats from butter, cream, full-fat cheese, and processed meat, and choosing olive oil, nuts, avocado, and fish instead. Without the variant, moderate fat intake is generally not a problem.

Frequently asked questions

Does this mean I can't eat any fat anymore?
No. This applies only to saturated fats and the amount you eat. Unsaturated fats from olive oil, nuts, and fish are recommended regardless of the variant.

Genes analyzed

APOA2

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Intense training causes oxidative stress in the muscle - this is part of the training stimulus, but it also needs to be cleared. Glutathione peroxidase and superoxide dismutase do exactly that. Genes partly determine how quickly this process happens, directly affecting the recovery time you need.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Overtraining doesn't come from training too hard, but from not getting enough recovery in between. If you know you need longer to recover, plan your week differently - and make more progress, not less.

What the variants mean

Certain variants are associated with a slower return of muscle damage markers to normal after intense exercise.

What you can do

If you recover more slowly, it helps to leave longer gaps between intense workouts, get enough sleep and protein, and do some light active recovery. High-dose antioxidant supplements are specifically not recommended here - they can blunt the training stimulus.

Frequently asked questions

How long should I rest between intense workouts?
As a rule of thumb, allow 48 hours before training the same muscle group again, or 72 hours if you recover more slowly. What matters is how strong and motivated you feel at your next workout.

Genes analyzed

GPX1SOD2

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Not everyone reacts to salt in the same way. In about a quarter to a third of the population, high salt intake causes a marked increase in blood pressure, while in others it has little effect. AGT encodes angiotensinogen, the starting material for the blood pressure-regulating renin-angiotensin system; CLCNKA affects salt reabsorption in the kidneys.

Genes analyzed2
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The blanket recommendation to reduce salt does not benefit everyone equally. For salt-sensitive people, it is one of the most effective measures, while for others it has little effect.

What the variants mean

Studies show that people with a salt-sensitive variant have a measurably greater rise in blood pressure with high sodium intake, and a correspondingly more pronounced decrease when intake is reduced.

What you can do

If you are particularly sensitive, it is worth looking at the main sources: bread, sausage, cheese, and ready meals make up most of your salt intake, not the salt shaker. Potassium from vegetables and fruit also helps lower blood pressure.

Frequently asked questions

How much salt is recommended?
The WHO recommends consuming less than 5 grams of table salt daily. In Germany, actual intake is significantly higher on average, mostly from processed foods.

Genes analyzed

AGTCLCNKA

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Free radicals are produced with every breath and every muscle contraction. The body has its own defense system for this: superoxide dismutase, catalase, glutathione peroxidase, and NQO1 neutralize these molecules in several stages. The effectiveness of this system varies genetically.

Genes analyzed4
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Oxidative stress is involved in cellular aging, recovery after exercise, and detoxification capacity. Those with a less effective system benefit more than average from an antioxidant-rich diet.

What the variants mean

Certain variants reduce the activity of protective enzymes. This becomes especially relevant under high stress - intense training, smoking, or heavy environmental pollution.

What you can do

Color on your plate is effective: berries, dark vegetables, green tea, nuts, and olive oil provide the substances that support the enzyme system. Important: High-dose antioxidant supplements can even reduce the training effect and are not recommended.

Frequently asked questions

Should I take antioxidant supplements?
Probably not. Studies show that high doses of individual antioxidants can dampen the training adaptation stimulus. This effect does not occur when they are obtained through food.

Genes analyzed

CATGPX1NQO1SOD2

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Selenoprotein P is the main transport protein for selenium and supplies it primarily to the brain and thyroid. Selenium is needed for enzymes that protect cells from oxidative stress, and for converting the thyroid hormone T4 into the active T3.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The soils in Central Europe are low in selenium, so intake from plant-based foods is correspondingly low. Those who also carry an unfavorable transport variant find it harder to achieve adequate levels.

What the variants mean

Certain variants are associated with lower selenium levels in the blood and affect how efficiently selenium reaches target organs.

What you can do

Good sources include fish, eggs, meat, and Brazil nuts—just a few a week are enough of the latter. The margin between adequate and excessive selenium intake is narrow, so supplementation should only be considered after testing.

Frequently asked questions

How many Brazil nuts a day are recommended?
One or two Brazil nuts a day are usually enough. Significantly more can already lead to excessive intake.

Genes analyzed

SEPP-1

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COMT breaks down dopamine in the frontal lobe, the area responsible for concentration, planning, and impulse control. The fast variant keeps dopamine levels low, while the slow variant keeps them high. This creates a trade-off: a high baseline level helps with concentration in calm situations, but under stress it can quickly become too much of a good thing.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The analysis explains an everyday difference: why some people thrive in exam situations while others freeze—even with the same preparation and ability.

What the variants mean

The variant known as Warrior breaks down dopamine quickly and is associated with better performance under pressure. The variant known as Thinker breaks it down slowly and is associated with better concentration and memory in calm situations, but with greater sensitivity to stress.

What you can do

With the Thinker variant, it’s worth scheduling challenging tasks for quiet periods, actively limiting stress peaks, and making sure to recover regularly. With the Warrior variant, clear deadlines and structure help, because too little stimulation is more likely to lead to underload.

Frequently asked questions

Is one variant better than the other?
No, it’s a trade-off. Both have advantages in different situations—which is why both have remained common in the population.

Does this say anything about my personality?
Just a very small part. Personality arises from thousands of gene variants, experiences, and environment. This one variant describes a tendency in the stress response, not character.

Genes analyzed

COMT

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Of all blood lipids, triglycerides respond most directly to diet—and genetic influence is also substantial. APOA5 and LPL regulate the breakdown of triglyceride-rich particles, while MLXIPL is a sugar sensor in the liver that switches on the production of new fat from carbohydrates.

Genes analyzed8
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

MLXIPL is the most interesting marker in this group: it explains why some people respond to a high-sugar diet with sharply rising triglycerides, while others barely respond. This turns a general recommendation into a targeted one.

What the variants mean

Certain variants are associated with considerably higher triglyceride levels on average, especially with high intake of sugar, fructose and alcohol.

What you can do

Avoiding alcohol and reducing sugar and refined carbohydrates have the quickest effect—triglycerides often respond within just a few weeks. Omega-3 fatty acids, weight loss and physical activity also help.

Frequently asked questions

Why are my triglycerides high even though I eat little fat?
Triglycerides are produced in the liver primarily from excess carbohydrates and alcohol, not from dietary fat. Sugar and alcohol are the more effective levers.

Genes analyzed

ANGPTL3APOA1APOA5FADS1GCKRLPLMLXIPLTRIB1

Related analysis topics

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Body weight is partly determined by heredity—to a considerable extent, with twin studies estimating the genetic contribution at 40 to 70 percent. This is not down to a single gene, but to the interplay of many variants, each with a small effect. The analysis includes FTO, the most extensively studied variant, as well as MC4R, TMEM18 and GNPDA2. Most of these genes do not affect metabolism, but the brain: they influence appetite, feelings of fullness and the reward response to food.

Genes analyzed8
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Knowing that their sense of fullness is genetically less pronounced helps people finally understand why portion control takes more effort for them than for others—and lets them choose targeted strategies that address the issue, instead of blaming themselves for a lack of discipline.

What the variants mean

Carriers of the FTO variant associated with increased risk weigh about 1.5 to 3 kilograms more on average than people without this variant. This is a statistical average across large groups, not a prediction for any individual. The key point is that physical activity has been shown to reduce this effect.

What you can do

With a pronounced predisposition, protein-rich meals, regular meal times, and mindful portion sizes are especially effective because they compensate for a weaker feeling of fullness. Studies also show that regular exercise significantly reduces the influence of the FTO variant—so predisposition is not destiny.

Frequently asked questions

Does an unfavorable variant mean I’ll inevitably gain weight?
No. These variants indicate a tendency, not a certainty. Their influence is much smaller than that of diet and exercise—and both can be used to offset it.

Why is FTO called the fat gene?
Because it was the first common variant whose link to body weight was conclusively proven. The name is misleading: the gene doesn’t make anyone fat; it mainly affects feelings of fullness.

Genes analyzed

BDNFFTOGNPDA2INSIG2MC4RNRXN3PCSK1TMEM18

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DRD2 encodes a dopamine receptor and is a key component of the reward system. GABRA influences the effects of the inhibitory neurotransmitter GABA, through which alcohol exerts its calming effect. Together, these variants shape how pleasurable alcohol feels.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The shift from enjoying alcohol to making it a habit happens gradually. Knowing you have a pronounced predisposition can help you set conscious limits earlier, rather than waiting until a pattern has formed.

What the variants mean

Certain variants are associated in studies with a stronger reward response to alcohol. The influence is real, but much smaller than that of environment, habits, and availability.

What you can do

Effective strategies include setting alcohol-free days, not keeping alcohol at home, and consciously replacing rituals. If you’re concerned about your drinking, seeking an assessment from a doctor or addiction counselor is the right approach—this analysis is no substitute.

Frequently asked questions

Does an unfavorable variant mean I will become addicted to alcohol?
No. This analysis does not predict any disease. It describes a tendency in the reward system that is far more strongly shaped by behavior and environment.

Genes analyzed

DRD2ERAP1GABRA

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FUT2 determines the so-called secretor status - whether certain sugar structures are produced on the mucous membrane of the intestine. These structures shape the composition of the gut flora and thereby indirectly affect vitamin B12 absorption. About 20 percent of Europeans are non-secretors.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

A B12 deficiency can develop unnoticed over years and leave nerve damage. People who are genetically less able to absorb it should check their levels regularly - especially if they eat little meat.

What the variants mean

Certain FUT2 variants are associated in studies with higher or lower B12 levels, depending on the specific variant. The association is mainly through the gut flora.

What you can do

For an unfavorable variant, checking holotranscobalamin in the blood is recommended, because this value measures the portion that is actually available for use. Supplementation is recommended anyway for a vegetarian or vegan diet.

Frequently asked questions

What is a non-secretor?
A person whose mucous membranes do not produce certain blood group sugar structures. This affects the gut flora and susceptibility to certain gastrointestinal infections - it is a normal variant, not a disease.

Genes analyzed

FUT2

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ALPL encodes alkaline phosphatase, an enzyme that processes the active form of vitamin B6 in the blood. Vitamin B6 is involved in over 100 enzyme reactions, particularly in protein metabolism and the production of signaling molecules such as serotonin and dopamine.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Higher turnover means higher requirements. Together with MTHFR and FUT2, this provides a picture of how well the overall methylation metabolism is supplied.

What the variants mean

Certain variants are associated with lower blood vitamin B6 levels, even when intake is not lower.

What you can do

Good sources include fish, poultry, potatoes, bananas, whole grains, and legumes. Long-term use of high-dose supplements is not recommended - persistently very high doses of B6 can cause nerve damage.

Frequently asked questions

Can you take too much vitamin B6?
Yes. Long-term use of high-dose supplements can cause abnormal sensations and nerve damage. It is not possible to overdose through diet.

Genes analyzed

ALPL

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SLC23A1 encodes a transporter that absorbs vitamin C from the intestine and prevents its loss through the kidneys. Vitamin C is needed for collagen production, iron absorption from plant sources, and as an antioxidant.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

With an unfavorable transport variant, vitamin C levels are lower for the same intake. This is especially relevant for connective tissue, wound healing, and iron metabolism.

What the variants mean

Studies show that carriers of certain variants have lower vitamin C levels in their blood, even though they consume similar amounts.

What you can do

Vitamin C is water-soluble and is not stored—the effect of several small servings throughout the day is better than one large serving. Good sources include bell peppers, broccoli, berries, and citrus fruits.

Frequently asked questions

Do high doses of vitamin C help more?
At around 200 milligrams per serving, the absorption rate drops significantly, and the excess is excreted. Several small servings are more effective than one high dose.

Genes analyzed

SLC23A1

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Vitamin D passes through several stages in the body, each involving one of the genes analyzed. DHCR7 controls the precursor in the skin, CYP2R1 the conversion in the liver, and GC produces the transport protein in the blood. Variants in these genes are among the strongest known genetic influences on vitamin D levels.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Vitamin D deficiency is already common in Germany because sunlight is insufficient from October to March. Those who also carry an unfavorable variant reach lower levels than others with the same amount of sun exposure and the same dose—and therefore need to pay closer attention.

What the variants mean

Studies show that carriers of certain variants have significantly lower average 25-OH vitamin D levels. This effect remains stable throughout the year and becomes stronger in winter.

What you can do

With an unfavorable variant, measuring blood 25-OH vitamin D levels is especially useful, ideally at the end of winter. The dosage should then be based on the measured level, not a general recommendation.

Frequently asked questions

Does genetic analysis replace blood testing?
No, it complements it. The genes indicate what your likely requirements are, while the blood test shows where your levels actually stand.

Do I need higher doses with an unfavorable variant?
Often, yes, but the specific amount should always be guided by measured blood levels and monitored by a doctor.

Genes analyzed

CYP2R1DHCR7GC

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Vitamin E is fat-soluble and is transported in the blood together with blood lipids. APOA5 regulates the breakdown of triglyceride-rich particles and thereby indirectly affects how much vitamin E circulates and reaches the tissues.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Vitamin E protects cell membranes and blood fats themselves from oxidation. The analysis links nutrient status and fat metabolism—two areas that are usually considered separately.

What the variants mean

Certain variants are associated with altered triglyceride levels and, as a result, different vitamin E levels.

What you can do

Good sources include vegetable oils, nuts, and seeds. Important: Vitamin E needs fat to be absorbed—nuts as a snack or oil in a salad are more effective than a supplement on an empty stomach.

Frequently asked questions

Are vitamin E supplements worthwhile?
Usually not with a balanced diet. Studies have shown no benefit from high-dose supplements taken individually.

Genes analyzed

APOA5

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TAS2R38 encodes a bitter taste receptor on the tongue. Depending on the variant, certain substances in cruciferous vegetables, chicory, arugula, grapefruit, and green tea taste strongly bitter, mildly bitter, or hardly bitter at all. People are broadly divided into non-tasters, regular tasters, and so-called supertasters.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

This analysis is the most convincing evidence that taste preferences are not learned. People who hated Brussels sprouts as children often simply had a different receptor—and need other ways to eat a vegetable-rich diet.

What the variants mean

Supertasters perceive bitter compounds much more intensely and, on average, eat fewer vegetables in studies. Non-tasters, on the other hand, more often choose more heavily seasoned and saltier foods.

What you can do

When bitterness is particularly strong, preparation can help: Roasting, caramelizing, and combining foods with fat or a little acidity noticeably reduces bitterness. Choosing milder varieties also makes a big difference.

Frequently asked questions

What is a supertaster?
A person who perceives bitter compounds more intensely than average. Depending on the population, this applies to about a quarter of people and is not a disorder, but a normal variation.

Genes analyzed

TAS2R38

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SLC2A2 encodes the glucose transporter GLUT2, which carries sugar in the liver and pancreas and also plays a role in sugar perception in the brain. People who perceive sweetness less strongly need more of it to get the same taste experience.

Genes analyzed1
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

The explanation is simple: It’s not the desire for sugar that differs, but the threshold at which something is perceived as sweet enough.

What the variants mean

Certain variants are associated with higher daily sugar intake, even though those affected don’t consider themselves particularly fond of sweets.

What you can do

A gradual reduction in sweetness over several weeks is effective. Taste perception adapts, and what used to taste normal then seems much too sweet.

Frequently asked questions

Do sweeteners help?
They replace calories but keep the sweetness threshold high. Anyone looking to recalibrate their perception has to gradually get used to less sweetness.

Genes analyzed

SLC2A2

Matching DNA test

WeightLoss | SLIM DNA Test

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Zinc is a component of over 300 enzymes and is essential for the immune system, wound healing, skin, and hormone production. The body does not build up large stores, so daily intake matters. The variants analyzed affect circulating zinc levels.

Genes analyzed3
Sample materialSaliva sample
Test requirementsNo special requirements
Validitylifelong, one-time analysis

Why this analysis matters

Zinc deficiency causes nonspecific symptoms such as frequent infections, poor wound healing, hair loss, and skin problems – issues rarely associated with zinc.

What the variants mean

Certain variants are associated with lower serum zinc levels, regardless of dietary intake.

What you can do

Good sources include meat, cheese, eggs, legumes, and pumpkin seeds. Phytates make plant-based zinc harder to absorb – soaking and fermenting significantly improve its availability. If you have an unfavorable variant, a blood test may be worthwhile.

Frequently asked questions

Does zinc affect testosterone levels?
A significant zinc deficiency can impair testosterone production. However, supplementation when there is no deficiency does not increase levels.

Genes analyzed

CA1NBDYPPCDC

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DNA analysis is for nutritional and lifestyle advice. It is not a diagnostic procedure, does not predict disease, and does not replace medical examination or advice. Genetic variants describe probabilities in population groups, not certainties for individuals.

Frequently asked questions

What should you know about DNA analyses?

What exactly is examined in a DNA analysis?

Depending on the test, between 33 and more than 170 gene variations, known as SNPs, are analyzed. These are individual points in the genome where people differ. They are linked to how your body processes nutrients, responds to training, or copes with stress. The glossary on this page lists the analysis topics these variants relate to.

What is the difference between a DNA analysis and a blood test?

DNA analysis shows your predispositions, while a blood test shows your current status. Your DNA doesn't change, so it describes what you're genetically predisposed to. Blood values change over the course of months and show how well-nourished you are right now. DNA explains more of the why, blood values the now, and together they provide more context than either one alone.

Do I need to repeat the DNA test at some point?

No, a DNA test is enough for a lifetime. Your DNA doesn’t change, so the result for the analyzed genetic variations stays the same. One saliva sample is enough, and your report remains valid. What can grow over time is our knowledge of individual variants, not your genetic makeup itself: your result stays the same; only our understanding of it grows.

Will the DNA analysis tell me whether I’ll get sick?

No. DNA analysis is intended for nutrition and lifestyle advice. It is not a diagnostic procedure, does not predict disease, and does not replace a medical examination or consultation. Genetic variants describe probabilities in population groups, not certainties for individuals. A predisposition is not a certainty, but one of several influences alongside diet, exercise, and daily life.

How does a DNA analysis work at mybody x?

Analysis takes 15 to 25 business days from the time your saliva sample arrives at the lab. You order the kit online, activate it at mylab.mybody-x.com, provide a saliva sample at home, and send it in the enclosed envelope. The sample is analyzed at a specialist laboratory in Germany, and you can download your report as a PDF from the customer portal.

What happens to my saliva sample and my data?

Your saliva sample and DNA sequence will be completely destroyed two months after the analysis is completed. Your sample is processed under a pseudonymous code, with the link to your identity stored separately. Your data is processed in accordance with ISO 27001 and the GDPR, and transmitted using SSL encryption. You can view your report in your customer portal at mylab.mybody-x.com.

Which DNA test is right for me?

For a single goal such as weight, exercise, nutrition, or skin, choose a DNA test focused on that specific topic; for a comprehensive overview, choose the analysis covering more than 170 genetic variations. The five DNA analyses range from 33 to over 170 genetic variations. If you're unsure, the test finder can help, or you can book a free consultation.

The DNA analysis is intended for nutrition and lifestyle advice. It is not a diagnostic procedure, does not predict disease, and does not replace a medical examination or consultation. Genetic variants describe probabilities in population groups, not certainties for individuals. The content is for general information and does not replace medical advice, diagnosis, or treatment.

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